According to new preprint by cardiovascular researcher Harriet Carroll and general practitioner Dr. Robin Kerr of Lund Universityblood coagulation in the form of “microcoagulation” is substantially responsible for the development of “long covid”.
Given that the symptoms that distinguish long covid are closely related to the “vaccine” injuries of the Wuhan coronavirus (Covid-19), Kerr and Carroll proposed that possibly the term “long covid” refers to people who received a covid vaccine and are now experiencing microcoagulation.
According to Etheresia Pretorius, a renowned research professor a University of Stellenboschand which focuses on the study of microcoagulation, although it may not be the only pathology among long-term covid patients, it is certainly one of the most prominent.
Clots begin to form in blood vessels when the contents of a vial of covid jab are injected and distributed throughout a person’s body, preventing nutrients from being exchanged. As a result, adjacent cells are deprived of oxygen and malfunction, which can lead to fatigue, shortness of breath, “brain fog” and even full-blown neuropathy.
“Some proteins involved in microcoagulation also play a role in mast cell activation and autoimmune pathways,” one report further explained. “Therefore, microcoagulation is associated with conditions such as autoimmunity and mast cell activation syndrome, characterized by allergic symptoms such as hives, swelling and difficulty breathing.”
Therapies that help restore circulation appear to be among the most successful in treating prolonged covid symptoms and microcoagulation, as covid jab spike proteins cause the production of larger than normal clots without thrombin and platelets. Dermatologist Dr. Angela Bowers says most of her chronically ill patients benefit greatly from these treatments.
The Frontline COVID-19 Critical Care Alliance (FLCCC) has published a series of documents explaining the pathology of blood clotting and some of the therapy procedures that this organization uses to help patients circulate. According to FLCCC experts, spike jab proteins are primarily to blame.
“Spike proteins are found on the surface of the COVID-19 virus, which uses them to invade cells and cause damage. Similarly, mRNA vaccines tell the body to produce spike proteins,” he explains the same report mentioned above.
Now that thrombin and platelets are absent, it is known that spike proteins can cause blood clots to form. This would explain why some of the dislodged clots we discussed above do not look like blood, but instead are a sticky, lighter-colored substance that ends up clogging people’s arteries.
“Spike proteins change the structure of beta and gamma fibrinogen, complement 3, and prothrombin, resulting in the development of blood clots that are larger and more difficult to break,” the reports state.
“Spike proteins damage the inner lining of blood vessels by binding to ACE-2 receptors on endothelial cells. ACE-2 has been identified as the primary receptor used by the SARS-CoV-2 virus for cellular entry, and endothelial cells are abundant in ACE-2 receptors, placing them particularly at risk of infection Consequently, spike proteins can enter cells endothelial cells and activate inflammatory pathways, which ultimately lead to the formation of blood clots.”
Clot formation that clogs the cardiovascular system is further triggered by spike proteins, which also cause hyperactivated platelets that are more likely to clump together and adhere to endothelial walls. Because these clots are so large and different, they don’t dissolve in the presence of trypsin, a digestive enzyme often used to dissolve other types of blood clots.
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